Title |
Inhibitory effects of fluorinated benzenesulfonamides on insulin fibrillation / |
Authors |
Hadi Ali Janvand, Saeid ; Ladefoged, Lucy Kate ; Zubrienė, Asta ; Sakalauskas, Andrius ; Christiansen, Gunna ; Dudutienė, Virginija ; Schiøtt, Birgit ; Matulis, Daumantas ; Smirnovas, Vytautas ; Otzen, Daniel E |
DOI |
10.1016/j.ijbiomac.2022.12.105 |
Full Text |
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Is Part of |
International journal of biological macromolecules.. Amsterdam : Elsevier. 2023, vol. 227, p. 590-600.. ISSN 0141-8130. eISSN 1879-0003 |
Keywords [eng] |
carbonic anhydrase inhibitors ; insulin ; protein fibrillation inhibitors ; sulfonamide compounds |
Abstract [eng] |
Amyloid fibrils are protein aggregates formed by protein assembly through cross β structures. Inhibition of amyloid fibril formation may contribute to therapy against amyloid-related disorders like Parkinson's, Alzheimer's, and type 2 diabetes. Here we report that several fluorinated sulfonamide compounds, previously shown to inhibit human carbonic anhydrase, also inhibit the fibrillation of different proteins. Using a range of spectroscopic, microscopic and chromatographic techniques, we found that the two fluorinated sulfonamide compounds completely inhibit insulin fibrillation over a period of 16 h and moderately suppress α-synuclein and Aβ fibrillation. In addition, these compounds decreased cell toxicity of insulin incubated under fibrillation-inducing conditions. We ascribe these effects to their ability to maintain insulin in the native monomeric state. Molecular dynamic simulations suggest that these compounds inhibit insulin self-association by interacting with residues at the dimer interface. This highlights the general anti-aggregative properties of aromatic sulfonamides and suggests that sulfonamide compounds which inhibit carbonic anhydrase activity may have potential as therapeutic agents against amyloid-related disorders. |
Published |
Amsterdam : Elsevier |
Type |
Journal article |
Language |
English |
Publication date |
2023 |
CC license |
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