| Abstract [eng] |
Chemical eye burns represent some of the most complex ophthalmological injuries, with potential to cause long-term visual impairment or blindness. While traditionally linked to damage in the anterior segment, research review indicates that the posterior segment — the retina and optic nerve — also sustains injury during burns. This study aims to evaluate the impact of chemical eye burns on the structure and function of the posterior segment, observe changes over various periods, and evaluate public awareness about these injuries to enhance education and prevention measures. Present research consists of two parts: a prospective clinical study and a public survey. Findings from the clinical study showed that patients with chemical eye burns, who had normal intraocular pressure, experienced thinning of the retinal nerve fiber and ganglion cell layers. This thinning continued for several months after the injury and was more severe with higher burn severity according to the Dua classification. The survey showed that the public has limited knowledge about chemical eye injuries, with more than half of the respondents showing low awareness. These findings confirm the posterior segment's key role in chemical burn pathogenesis, emphasizing the need for neuroprotective strategies and public education to prevent vision loss. |
